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Facial paralysis ischemic event pdf

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An ischemic facial paralysis was induced by injecting a vasoconstrictor into the facial canal in dogs sensitized to horse serum. On histopathologic section the nerve changes were similar to the changes known to occur in patients with Bell's palsy. The paralysis of Bell's palsy may be the result of ischemia and compression of the facial nerve within the fallopian canal in individuals showing a response of hypersensitivity to an active pharmocological agent acting locally. Arch Otolaryngol. Coronavirus Resource Center.
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A Focal Pontine Infarct Presenting as Unilateral Facial Nerve Paralysis

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Management of peripheral facial nerve palsy | SpringerLink

Introduction: The ethiologic diferencial diagnostic for facial nerve paralisis is still a challenge and the literature has shown conflictive results concerning its epidemiology. Objective: To outline the incidence of the different ethiologies and the profile of peripheral facial nerve paralysis patients in the otolaryngology ambulatory of the Faculdade de Ciencias Medicas e Biologicas da PUC-SP - campus Sorocaba. Method: The records of 54 patients with facial nerve paralysis seen during the years of and were analysed retrospectively. There is an equilibrium concerning to the gender, with a slight prevalence for males and for the right side of the face. Its incidence ranges from 20 to 30 cases per
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Management of peripheral facial nerve palsy

Three quarters of peripheral FNP are primary and one quarter secondary. The most prevalent causes of secondary FNP are systemic viral infections, trauma, surgery, diabetes, local infections, tumor, immunological disorders, or drugs. The diagnosis of FNP relies upon the presence of typical symptoms and signs, blood chemical investigations, cerebro-spinal-fluid-investigations, X-ray of the scull and mastoid, cerebral MRI, or nerve conduction studies. Treatment of secondary FNP is based on the therapy of the underlying disorder.
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Brainstem infarction typically presents with vague symptoms, including headache, nausea, vomiting, and vertigo. Her atypical presentation of unilateral facial nerve paralysis in the context of nausea, vomiting, and vertigo prompted neurological studies, which were significant for a small punctate infarct in the pons involving the right facial colliculus. Stroke and other clinically emergent etiologies should be considered high on the differential diagnosis when patients have neurological signs and symptoms in addition to facial nerve palsy. The symptoms are often vague, including dizziness, visual-field disturbances, nausea, and vomiting [ 1 ]. Rapid diagnosis and treatment is essential for a full recovery.
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